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The mechanism of HLA-DM induced peptide exchange in the MHC class II antigen presentation pathway. Achondrogenesis type II (Langer-Saldino), caused by a genetic defect in the major cartilage matrix protein, collagen type II, is a rare and severe skeletal dysplasia. The combined approach led to an estimate of a potentially effective dose for BAY 60-5521 of 51 mg in humans. SLM treatment with myelobromol inhibited both cytotoxic responses. Our conclusion from this analysis is that the most important properties of NESs are accessibility and flexibility allowing relevant proteins to interact with the signal. Cardiovascular disturbances were reduced and fluctuations in plasma catecholamines were decreased.
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